Some of My Little Girl Outfits (and Stories)

tobid03 said:
Thanks for all that info, but the bedwetting part is just a part of that little girl fantasy.

On another note, I got some red marks from that gymnastics leotard. Might have to try a size larger. I hate being sensitive to pressure urticaria (welts from pressure that can leave marks for a long time).
They are made for a woman’s body. The shoulder to crotch measurement is to small for most men.
 
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tobid03 said:
I hate being sensitive to pressure urticaria (welts from pressure that can leave marks for a long time).
I'm sorry you have this condition. More information can be found.

Pressure Urticaria


Background

Pressure urticaria is an uncommon form of physical urticaria, a subset of chronic urticaria, which presents with erythematous swelling at sites of pressure. Chronic urticaria is termed when patients have ongoing urticaria for more than 6 weeks. An inciting event or etiology is usually not identified for patients with chronic urticaria—hence the term chronic idiopathic urticaria (CIU) is often used. A proportion of patients diagnosed with chronic urticaria have physical urticaria, also referred to as chronic inducible urticaria (CIndU), which is urticaria incited by a physical stimulus, such as mechanical (friction, vibration, pressure) urticaria, thermal (heat or cold) urticaria, solar urticaria, and symptomatic dermatographism.

Pressure urticaria may occur immediately (within minutes) or, more commonly, 4-6 hours after a pressure stimulus. For this reason, the term delayed pressure urticaria (DPU) is typically used. It appears as an erythematous, cutaneous, and often subcutaneous edema. The reaction may last up to 72 hours and can be associated with pruritus, burning, and pain. Pressure sites, including the hands, feet, trunk, buttocks, and legs, are most commonly affected. Lesions can be induced by a variety of stimuli, including standing, walking, wearing of tight clothes, and sitting or
leaning on a hard surface. See the image below.

Delayed pressure urticaria. Courtesy of DermNet NeDelayed pressure urticaria.


See emedicine.medscape.com/article/1050387-overview?reg=1
 
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Pressure Urticaria

Pathophysiology

The pathogenesis of DPU (delayed pressure urticaria) is relatively unknown. Although the trigger stimulus of pressure is identified, no allergen has been established. Urticaria (hives) is a type I hypersensitivity reaction, where autoantibodies against IgE molecules are involved. When an antibody or autoantibody binds to the IgE molecule, a bridge is formed between two or more IgE molecules. This induces mast cell degranulation, releasing multiple proinflammatory mediators, including histamine, leukotriene, and prostaglandin. In CIU (chronic idiopathic urticaria), the mast cells are inappropriately activated. Histamine levels are increased in the lesional skin, while intracellular histamine levels are decreased in peripheral white blood cells. There is also an increased stimulation of histamine release. Despite these findings, histamine is unlikely to be the sole mediator in pressure urticaria. This is further demonstrated in the inconsistent effectiveness of antihistamine treatment in pressure urticaria.

Other mediators are believed to be involved, and mast cells may be triggered by IgE-independent pathways. Patients with ClndU (chronic inducible urticaria) have increased serum levels of IgE. This includes eosinophils (as suggested by the presence of eosinophilia), ECP (eosinophil cationic protein (ECP), and ECF (eosinophil cationic factor) found in biopsy specimens from select patients with DPU, particularly those with bullous DPU. In addition, elevated concentrations of interleukin (IL)–1a, IL-5, and IL-6; TNF (tumor necrosis factor)–alpha; and leukotrienes have also been found in lesional skin of pressure urticaria patients. Vascular endothelial growth factor has also been found to be elevated in patients with DPU. Abnormalities in platelets and fibrin or fibrinolysis have also been investigated. Systemic inflammation has also been suggested, with elevations seen in CRP (C-reactive protein) and sCD40L, a platelet activator.

Etiology

Pressure stimuli may include the following:
  • Standing, walking, leaning, or sitting
  • Using tools
  • Carrying a handbag or backpack
  • Wearing tight-fitting clothes (e.g., bra straps or watches)

    Occasionally, DPU (delayed pressure urticaria) is aggravated by heat, aspirin, or menstruation. Exacerbation of the condition during medical procedures is a reasonable possibility; urticaria flares following endoscopy have been described.

    Epidemiology​

    DPU is generally considered a rare entity; however, this may be because it is not consistently recognized. Approximately 37% of patients with chronic spontaneous urticaria also have pressure urticaria. The mean age of onset of DPU is in the 30s (range, 5-63 y). DPU is slightly more common in men than in women.

    Prognosis​

    DPU is a chronic disease that can last for years (mean, 9 y; range, 1-40 y). The morbidity of DPU varies, depending on the severity and the response to treatment. In some patients, this condition can be disabling, especially in patients who perform manual labor.

    QOL (quality-of-life) tools have demonstrated that patients with urticaria can show impairments in QOL scores similar to those seen in patients with chronic dermatoses such as psoriasis and atopic eczema. QOL scores were lowest for patients with CIU (chronic idiopathic urticaria) compared with psoriasis and atopic dermatitis for “self-perception,” “social functioning,” and “treatment-induced restrictions.”

    Clinical Presentation​

    History and Physical Examination​

    The clinical manifestations of DPU differ from those typical of most types of urticaria. Onset is typically delayed, most commonly occurring 4 hours after the pressure stimulus. Less commonly, wheals due to pressure develop within minutes, in which case they may be confused with dermatographism. The lesions of DPU can persist for several hours and sometimes for as long as 72 hours, unlike those in typical urticaria, which resolve within 24 hours. The physical findings in DPU include wheals, typically involving the palms, soles, legs, and waist. DPU lesions may also involve the genitals. The wheals, which appear as deep dermal and subcutaneous swellings, often resemble angioedema more than they do typical urticaria. Typical urticaria may also be present as a result of coexisting CIU (chronic idiopathic urticaria) or some other chronic physical urticaria. The lesions may be pruritic, painful, or burning. They can occur on any cutaneous surface and may mimic angioedema. With severe episodes, patients may experience fever, malaise, fatigue, chills, headache, and generalized arthralgias. Affected areas can be refractory to the development of new lesions for 1-2 days. As many as 60% of individuals with DPU have concomitant CIU, immediate or delayed dermographism (dermatographism), or angioedema. In some reports, the incidence of DPU in patients with CIU varies considerably, ranging from 2% to 40%; other reports estimate the rate to be 2-4%.


    Diagnostic Considerations​

    The development of systemic signs of angioedema or urticaria, such as severe swelling and shortness of breath, may coexist with DPU. In addition to the conditions listed in the differential diagnosis, other problems to be considered include delayed dermographism and vibratory urticaria.

    Differential Diagnoses​

    Workup​

    Laboratory Studies​

    An elevated WBC (white blood cell) count or neutrophilia may be present. Complement levels are normal.
    Some patients with DPU also have concomitant CIU. Basic testing for CIU includes CBC count with differential, erythrocyte sedimentation rate, and/or C-reactive protein. Extended diagnostic testing includes the following.
    • Thyroid function tests and antibody testing for autoimmune thyroid disease
    • Antibody testing for Helicobacter pylori
    • Autoantibody to IgE

      Allergy testing (skin tests and/or allergen avoidance tests)

      Pressure Challenge Testing​

      Pressure challenge testing (with the dermographometer or the suspended-weight method) may be performed for DPU. Because therapy may influence test results, it is recommended that testing be performed at a time when therapy has been interrupted or stopped for at least 3 days; however, some patients may have more severe disease that does not allow this. Repetitive testing can be used to assess response to therapy.

      Multiple methods of applying measured amounts of pressure can be used to test for the development of DPU. A consensus conference review suggests using the following approaches :
      shoulder strap 3 cm wide with a 7-kg weight for 15 minutes
      rod 1.5 cm in diameter with a 2.5-kg weight for 15 minutes
      rod 6.5 cm in diameter with a 5-kg weight for 15 minutes
      dermographometer to a pressure of 100 g/mm2 for 70 seconds

      The tests are most often applied to the shoulders, upper back, posterior thighs, or volar forearm. Pressure provocation tests should be read at 6 hours. Both the weight and the application time should be recorded. The duration of application is inversely related to the pressure applied; for example, wheals take longer to develop with a lower pressure than with a higher pressure.

      Histologic Findings​

      The histologic features of DPU lesions are variable, often depending on the age of the lesion. Biopsy within hours demonstrates moderate-to-heavy infiltration of eosinophils with neutrophils and lymphocytes in a perivascular and interstitial pattern in the dermis and subcutaneous fat. Degranulated mast cells may be noted. Biopsy of an older DPU lesion (>24 h) demonstrates eosinophils and lymphocytes. No vessel-related changes (e.g., leukocytoclasia or fibrinoid necrosis), such as those noted in urticarial vasculitis, are seen. Direct immunofluorescence test results are negative. Several reports of bullous pressure urticaria have been reported, and histologic findings show spongiosis and intraepidermal bullae associated with an eosinophil-rich inflammatory infiltrate in the superficial and deep dermis.

      Treatment & Management​

      Approach Considerations​

      Patients should avoid triggers of urticaria and attempt to limit pressure stimuli. A simple intervention is to broaden the handles on heavy items or straps on clothing to disperse the pressure over a larger area. However, avoidance is not easy and may not be helpful in patients with moderate-to-severe disease. Second-generation antihistamines are considered first-line treatment for DPU; however, DPU is relatively refractory to antihistamines. Omalizumab has shown promise for CindU (chronic inducible urticaria) in patients with disease refractory to antihistamines, with few adverse effects. Systemic corticosteroid treatment leads to improvement of DPU; however, its use is limited owing to its adverse effect profile. Second- and third-line agents used have included colchicine, dapsone, sulfasalazine, montelukast, chloroquine, cyclosporine, IVIg (intravenous immunoglobulin), and anakinra. Restrictions in activity depend on the severity of the disease. Consult a dermatologist or allergist for evaluation for other causes of urticaria.




      Pharmacologic Therapy​

      Antihistamines can reduce the severity of swelling and frequency of urticaria and are helpful in controlling associated CIU. There are few adverse effects of antihistamines, mainly sedation, which is less potent in the second-generation antihistamines. Some authors have suggested up to 4 times the recommended dose of nonsedating antihistamines to achieve control. However, DPU has notoriously been known to be refractory to antihistamines and antihistamines may not control the symptoms completely. Omalizumab, a recombinant DNA monoclonal antibody that binds to IgE, is showing promising results in CIU, with few adverse effects, owing to its ability to decrease mast cell degranulation. It is approved by the US Food and Drug Administration for CIU, but not necessarily for CIndU. Studies published in early 2019 express hope there will soon be approval for CIndU. Three phase 3 clinical trials, ASTERIA I, ASTERIA II, and GLACIA, involving over 900 patients with chronic spontaneous urticaria showed the benefits of omalizumab. A phase 3, multicenter study has shown that omalizumab decreased itchiness and hives and increased QOL in patients with CIU or chronic spontaneous urticaria, whose disease had been refractory to antihistamine therapy. Patients, including those with DPU, who initially had a positive response to omalizumab, and then relapsed after stopping treatment, achieved remission after restarting omalizumab. Multiple case reports have reported the benefit of omalizumab treatment in patients with DPU.

      Steroids are best restricted for recalcitrant and severe DPU. Prednisone has some clinical efficacy, but long-term therapy is problematic because of its many adverse effects. One study of a small group of patients found high-potency topical steroids to be efficacious for reducing edema, erythema, and pruritus associated with DPU lesions. Patients who see improvement with systemic steroid therapy often relapse when these agents are discontinued. The adverse effects of steroids must also be considered and managed. Methotrexate has been used successfully in steroid reduction in a few patients with steroid dependent DPU. NSAIDs produce variable responses. As treatment, they may be suboptimal because they, along with aspirin, may worsen urticaria and angioedema.

      Other therapeutic agents that have been tried including colchicine, dapsone, sulfasalazine, and montelukast. Colchicine has been largely ineffective as a therapy. Dapsone has demonstrated beneficial results persisting after treatment in a small study. A 2015 case series showed that in 17 patients treated with sulfasalazine, 11 had complete or near complete resolution and four had a partial response. Reports from small studies have found leukotriene antagonists, alone or in combination, to be efficacious for the treatment of DPU; other forms of chronic urticaria have not demonstrated similar responses to this treatment. Case reports have demonstrated some success with chloroquine, cyclosporine, IVIg, tricyclic antidepressants, selective serotonin reuptake inhibitors, and anakinra. Combination therapy may decrease disease activity. Adjunctive agents that reportedly have been successfully used in this context include leukotriene antagonists (e.g., montelukast, zafirlukast) and H2-receptor antagonists (e.g., famotidine, ranitidine).

      Medication Summary​

      The goals of pharmacotherapy are to reduce morbidity and to prevent complications. The mainstay agents used in the management of pressure urticaria include antihistamines and corticosteroids. The benefit of omalizumab has been established in CIU and shows benefit for patients with DPU. Other medications such as leukotriene antagonists, dapsone, sulfasalazine, methotrexate, cyclosporine, and intravenous immunoglobin (IVIg) have limited data, with only few case reports.

      Antihistamines, 2nd Generation​

      Class Summary​

      Antihistamines may be useful in helping control symptoms of chronic urticaria, which frequently coexists with DPU. Second-generation antihistamines, also known as less-sedating or low-sedation antihistamines, produce less sedation than traditional H1 blockers because they are less lipid-soluble and only cross the blood-brain barrier in small amounts. They also have longer half-lives, allowing less frequent dosing. Many H1 antagonists are metabolized through the cytochrome P-450 system. Important exceptions include cetirizine, levocetirizine, and fexofenadine.

      Fexofenadine (Allegra, Allegra Allergy 12 Hour, Allegra Allergy 24 Hour)​


      Fexofenadine is a nonsedating second-generation medication that has fewer adverse effects than first-generation medications. It competes with histamine for H1 receptors in the gastrointestinal (GI) tract, blood vessels, and the respiratory tract, reducing hypersensitivity reactions. Fexofenadine does not sedate. It is available in once-daily and twice-daily preparations.

      Cetirizine (Aller-Tec, Children's Zyrtec Allergy, Children's Zyrtec Hives Relief)​

      Cetirizine selectively inhibits H1 receptor sites in blood vessels, the GI tract, and the respiratory tract, thereby inhibiting the physiologic effects that histamine normally induces at H1 receptor sites. Once-daily dosing is convenient; bedtime dosing may be useful if sedation is a problem.

      Loratadine (Claritin, Claritin RediTabs, QlearQuil All Day & All Night 24 Hour Allergy Relief)​


      Loratadine selectively inhibits peripheral histamine H1 receptors.

      Levocetirizine (Xyzal)​


      Levocetirizine is an H1-receptor antagonist and an active enantiomer of cetirizine. Peak plasma levels are reached within 1 hour, and the half-life is approximately 8 hours. Levocetirizine is available as a 5-mg breakable (scored) tablet and a 0.5 mg/mL oral solution. It is indicated for uncomplicated skin manifestations of CIU.

      Desloratadine (Clarinex, Clarinex RediTabs)​


      Desloratadine is a long-acting tricyclic histamine antagonist that is selective for H1 receptors. It is a major metabolite of loratadine, which, after ingestion, is extensively metabolized to the active metabolite 3-hydroxydesloratadine.

      diphenhydramine (Rx, OTC)​

      Brand and Other Names: Benadryl, Benadryl Allergy Dye-Free LiquiGels, more...

      Dosing & Uses​

      Dosage Forms & Strengths​

      oral liquid​

      • 12.5mg/5mL (Benadryl Allergy Childrens, PediaCare Children’s Allergy, Allergy Relief Childrens)
      • 50mg/30mL (ZzzQuil)

      elixir​

      • 12.5 mg/5mL (Altaryl)

      syrup​

      • 12.5 mg/5mL (Altaryl, Quenalin, Silphen Cough)

      tablet​

      • 25mg (Benadryl, Nytol, Simply Sleep, Sominex, Simply Allergy, Tetra-Formula Nighttime Sleep)
      • 50mg (Aler-Dryl, Nytol Maximum Strength)

      capsule​

      • 25mg (Benadryl Allergy Dye-Free Allergy, Anti-Hist Allergy, Allergy Relief, Diphenhist, Geri-Dryl)
      • 50mg (Banophen, Pharbedryl)

      tablet, chewable​

      • 12.5mg (Benadryl Allergy Childrens)

      tablet, dispersible​

      • 25mg (Unisom SleepMelts)

      strip​

      • 12.5mg (Triaminnic Cough/Runny Nose)

      injectable solution​

      • 50mg/mL

      Allergic Reaction​

      25-50 mg PO q6-8hr; not to exceed 300 mg/day
      10-50 mg (no more than 100 mg) IV/IM q4-6hr; not to exceed 400 mg/day

      Insomnia​

      50 mg PO 30 minutes before bedtime

      Cough​

      25-50 mg PO q4hr PRN (syrup preferred); not to exceed 150 mg/day

      Motion Sickness​

      Treatment or prophylaxis: 25-50 mg PO q6-8 hr
      Alternatively, 10-50 mg/dose for treatment; may use up to 100 mg if needed; not to exceed 400 mg

      Parkinsonism​

      25 mg PO q8hr initially, then 50 mg PO q6hr; not to exceed 300 mg/day
      Alternatively, 10-50 mg IV at a rate not to exceed 25 mg/min; not to exceed 400 mg/day; may also administer 100 mg IM required





 
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Seasonedcitizen said:
They are made for a woman’s body. The shoulder to crotch measurement is to small for most men.
Thanks! Would you happen to know how many sizes to go up?
 
tobid03 said:
I also got these ruffle socks. I just love how little-girlish the ruffles are. Now I just need the perfect shoes to match.
View attachment 96468
Mary Jane sandals are perfect with pretty socks like those.
 
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tobid03 said:
On another note, I got some red marks from that gymnastics leotard. Might have to try a size larger. I hate being sensitive to pressure urticaria (welts from pressure that can leave marks for a long time).
I got other sizes of a gymnastics leotard. Even though they’re technically too big according to gymnastics regulations (but still kind of fit), I’m still having the same issue.
 
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I figured out how to wear my gymnastics leotard without getting welts on my shoulder from it being too tight. I tried hanging it wet after washing with canned food at the bottom to stretch it, and it worked!

I love wearing it when I feel like being a little girl or being forced to be a little girl while exercising.
 
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realy, realy cute. So much fun to wear.
 
tobid03 said:
I figured out how to wear my gymnastics leotard without getting welts on my shoulder from it being too tight. I tried hanging it wet after washing with canned food at the bottom to stretch it, and it worked!

I love wearing it when I feel like being a little girl or being forced to be a little girl while exercising.
I do that with my cotton 4Care bodysuits. Just enough stretch.
 
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LittleMaya said:
Your choices for nightgowns are absolutely adorable... you have fab taste. Although I have a few, and a girls size 18 will usually do the trick for me, it's the length I struggle with. A girls nightgown is designed to be close to, or below the knee, and I'm just way too tall... they end up just covering my butt. Same prob with jammie bottoms.. they're just too darn short. I'd buy that blue princess one in a heartbeat if it was actually long enough. :cry:
As for the car seat: I **love** my carseat.. but it may never actually get used in a car because being 5'12" tall, there's never enough headroom for being in it.
I love a nightie that only just covers my bum. It means my nappy and baby pants are on display.
 
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tobid03 said:
I figured out how to wear my gymnastics leotard without getting welts on my shoulder from it being too tight. I tried hanging it wet after washing with canned food at the bottom to stretch it, and it worked!

I love wearing it when I feel like being a little girl or being forced to be a little girl while exercising.
I love wearing a leotard and tights over a nappy and plastic baby pants.

The leotard holds my nappy nice and snug to my bum, even when I’ve wet, and with the tights under my leotard there’s no chance of being able to use a toilet, so I have to just use my nappy.
 
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Hi Ali , I dress exactly the same! As you said no option but to wet your nappy! I just love being a little girl!
 
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tobid03 said:
I figured out how to wear my gymnastics leotard without getting welts on my shoulder from it being too tight. I tried hanging it wet after washing with canned food at the bottom to stretch it, and it worked!

I love wearing it when I feel like being a little girl or being forced to be a little girl while exercising.
What kind of leotard is it? Iis it sleeveless, short-sleeved or long-sleeved? Is it cotton or a material like spandex? I'm only asking because I love wearing women's leotards and catsuits, and in the process of building a collection/wardrobe of them.
 
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very nice, love them
 
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Sissyleslie said:
What kind of leotard is it? Iis it sleeveless, short-sleeved or long-sleeved? Is it cotton or a material like spandex? I'm only asking because I love wearing women's leotards and catsuits, and in the process of building a collection/wardrobe of them.
It’s a sleeveless shiny spandex/polyester mix made by GK.
 
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Ali123 said:
I love wearing a leotard and tights over a nappy and plastic baby pants.

The leotard holds my nappy nice and snug to my bum, even when I’ve wet, and with the tights under my leotard there’s no chance of being able to use a toilet, so I have to just use my nappy.
I like this too with my ballet clothes. I like to imagine I got turned into a little girl around potty training age. I'm trying to prove my maturity but having to rush to the bathroom, take off possibly my ballet wings as well as my leotard, tights, and diaper prove too much for my toddler body, and I wet my diaper.
 
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tobid03 said:
It’s a sleeveless shiny spandex/polyester mix made by GK.
Sounds very nice. I only have one sleeveless leotard- a light blue one. It is similar to what the main character wears in the Belgian film 'Girl '.
 
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Sissyleslie said:
Sounds very nice. I only have one sleeveless leotard- a light blue one. It is similar to what the main character wears in the Belgian film 'Girl '.
Loved that Film
 
Got a few more things that I plan to post as I have time. Got this girls butterfly pajamas set specifically for the butterfly pjs. It's a perfect replica of little girls pajamas. Makes me feel obviously like a little girl with it on. What else could I be? I do wish the striped pants were something else or like a solid light red or pink. I don't really care for the flower all-over print one though, but I do love the "ruffle" edging on both pants leg cuffs.


1704092284781.png
 
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tobid03 said:
I also got these ruffle socks. I just love how little-girlish the ruffles are. Now I just need the perfect shoes to match.
View attachment 96468
Mary Janes would be the answer - pastel (think-pink) sneakers - light colored ankle boots (low cut to reveal the frilly top)
 
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